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The Na+, K+ pumps keep us going : how passive and active transport of sodium (Na+) and potassium (K+) control performance and fatigue in skeletal muscle / by Torben Clausen.

By: Contributor(s): Material type: TextTextPublisher: Aarhus, Denmark : Aarhus University Press, 2016Description: 1 online resource (120 pages) : illustrations (some color), charts (some colorContent type:
  • text
Media type:
  • computer
Carrier type:
  • online resource
ISBN:
  • 9788771842470 (electronic bk)
  • 8771842470 (electronic bk)
Subject(s): Genre/Form: Additional physical formats: Print version:: No titleDDC classification:
  • 572.793 23
LOC classification:
  • QP609.S63 C53 2016
Online resources:
Contents:
The Na+, K+ pump and its discovery -- The transport and distribution of Na+ and K+ in skeletal muscle and how they are quantified -- Rapid passive fluxes of Na+ and K+ start muscle contraction and set a limit to excitability -- The definition, structure and function of the Na+, K+-pump -- The synthesis and localization of the Na+, K+ pumps in skeletal muscle -- Measurements of the content of [3H]ouabain binding sites indicate that in skeletal muscle, the major part of these binding sites of the Na+, K+ pumps are localized on the outer surface of sarcolemma and in the t-tubules -- The content of Na+, K+ pumps in skeletal muscles and how it can be quantified -- Muscle contractions, Na+, K+ transport and sodium potassium fatigue -- The rate of [³H]ouabain binding to sarcolemma can be quantified and what information may this provide? -- Measurements of [3H]ouabain binding to other cell types and preparations -- Acute and long-term regulation of Na+, K+ pumps in skeletal muscle -- Catecholamines and caffeine -- Peptide hormones stimulating the Na+, K+ pumps -- Insulin-like growth Factore-I (IGF-I) -- CGRP and other calcitonins -- Amylin, related peptides, and other stimuli for the Na+, K+ -pumps -- The importance of Na+-influx in causing depolarization and muscle fatigue -- Effect of varying Na+, K+-pump stimulating agents on intracellular Na+ in rat soleus muscles and how this may compensate functional defects caused by plasma membrane leakage -- Long-Term regulation of Na+, K+ pump content -- Training, inactivity and denervation -- Muscular dystrophy and McArdle disease -- Hyperkalemic periodic paralysis in horses, human subjects and mutant mice with a similar geneti anomaly -- K+ deficiency and K+ restoration -- Thyroid hormones ans starvation -- Thermogenic actions of thyroid hormones and malignant hyperthermia (MH) -- Diabetes -- Steroid hormones, glucocorticoids, aldosterone -- Major conclusions and general perspectives.
Holdings
Item type Current library Collection Call number Status Date due Barcode Item holds
eBook eBook e-Library EBSCO Medical Available
Total holds: 0

Includes bibliographical references (pages 107-120)

The Na+, K+ pump and its discovery -- The transport and distribution of Na+ and K+ in skeletal muscle and how they are quantified -- Rapid passive fluxes of Na+ and K+ start muscle contraction and set a limit to excitability -- The definition, structure and function of the Na+, K+-pump -- The synthesis and localization of the Na+, K+ pumps in skeletal muscle -- Measurements of the content of [3H]ouabain binding sites indicate that in skeletal muscle, the major part of these binding sites of the Na+, K+ pumps are localized on the outer surface of sarcolemma and in the t-tubules -- The content of Na+, K+ pumps in skeletal muscles and how it can be quantified -- Muscle contractions, Na+, K+ transport and sodium potassium fatigue -- The rate of [³H]ouabain binding to sarcolemma can be quantified and what information may this provide? -- Measurements of [3H]ouabain binding to other cell types and preparations -- Acute and long-term regulation of Na+, K+ pumps in skeletal muscle -- Catecholamines and caffeine -- Peptide hormones stimulating the Na+, K+ pumps -- Insulin-like growth Factore-I (IGF-I) -- CGRP and other calcitonins -- Amylin, related peptides, and other stimuli for the Na+, K+ -pumps -- The importance of Na+-influx in causing depolarization and muscle fatigue -- Effect of varying Na+, K+-pump stimulating agents on intracellular Na+ in rat soleus muscles and how this may compensate functional defects caused by plasma membrane leakage -- Long-Term regulation of Na+, K+ pump content -- Training, inactivity and denervation -- Muscular dystrophy and McArdle disease -- Hyperkalemic periodic paralysis in horses, human subjects and mutant mice with a similar geneti anomaly -- K+ deficiency and K+ restoration -- Thyroid hormones ans starvation -- Thermogenic actions of thyroid hormones and malignant hyperthermia (MH) -- Diabetes -- Steroid hormones, glucocorticoids, aldosterone -- Major conclusions and general perspectives.

Description based on print version record

Added to collection customer.56279.3

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