TY - BOOK AU - Wasterlain,Claude G. AU - Treiman,David M. TI - Status epilepticus: mechanisms and management SN - 9780262285834 AV - RC372 .S769 2006eb U1 - 616.8/53 22 PY - 2006/// KW - Epilepsy KW - Convulsions KW - Treatment KW - Brain KW - Diseases KW - Central nervous system KW - Nervous system KW - Status Epilepticus KW - Brain Diseases KW - Central Nervous System Diseases KW - Nervous System Diseases KW - Disease KW - Seizures KW - Traitement KW - État de mal épileptique KW - Épilepsie KW - Cerveau KW - Maladies KW - Système nerveux central KW - Système nerveux KW - disease KW - aat KW - illness KW - PSYCHOLOGY KW - Neuropsychology KW - bisacsh KW - fast KW - Epilepsie KW - gtt KW - Behandeling KW - NEUROSCIENCE/General KW - Electronic books KW - Review N1 - Includes bibliographical references and index; History and methods --; Cognitive domains --; Developmental and clinical applications; Electronic reproduction; [Place of publication not identified]; HathiTrust Digital Library; 2010 N2 - "Interest in status epilepticus--the most extreme form of epilepsy, involving continuous seizures--has surged in the last 20 years. Since 1979 there have been over 4,000 publications on the subject, including more than 1,700 in the last five years. No other text provides such a comprehensive review of the recent advances in the field of status epilepticus. The book focuses on the two areas in which progress has been most rapid: basic mechanisms and treatment. There is now a greater understanding of the mechanisms and complications of status epilepticus at the molecular level, which should eventually lead to improved therapy, and treatment strategies today have a greater sense of urgency because of the realization that neuronal apoptosis and necrosis can be triggered very quickly. After an overview of history, classification, and epidemiology, the contributors consider clinical phenomenology, biological markers, pathophysiology, brain damage, epileptogenesis, therapeutic principles, pharmacology, and therapeutic management. Their contributions are equally divided between studies of basic mechanisms in animal models and clinical studies, so that the reader can turn easily from the reductionist experiment that isolates a small component of status to the complex clinical situation in which these principles can translate into therapeutic action. The goal is to provide a scientific rationale for clinical decisions while developing therapeutic attitudes that are firmly grounded in pathophysiology"--MIT CogNet UR - https://search.ebscohost.com/login.aspx?direct=true&scope=site&db=nlebk&db=nlabk&AN=156965 ER -